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TLR4-dependent fibroblast activation drives persistent organ fibrosis in skin and lung.

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TLR4-dependent fibroblast activation drives persistent organ fibrosis in skin and lung.

 

The results suggest that systemic scleroderma patients with high TLR4 activity might show optimal therapeutic response to selective inhibitors of MD2/TLR4 complex formation. PubMed, JCI Insight, 2018 Jul 12;3(13). (Also see Fibroblasts, Skin Fibrosis and Pulmonary Fibrosis Research)

 

This item was posted in the ISN Newsroom. Please check the newsroom daily for updates on scleroderma and other related articles.


Jo Frowde

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International Scleroderma Network (ISN)

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